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	<title>Cardiophile Electrophysiology</title>
	<atom:link href="http://www.cardiophile.net/feed" rel="self" type="application/rss+xml" />
	<link>http://www.cardiophile.net</link>
	<description>Blog devoted to Cardiac Electrophysiology Fellows</description>
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			<item>
		<title>Osler&#8217;s sign</title>
		<link>http://www.cardiophile.net/2009/09/oslers-sign.html</link>
		<comments>http://www.cardiophile.net/2009/09/oslers-sign.html#comments</comments>
		<pubDate>Thu, 24 Sep 2009 15:44:46 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[Osler's sign is the palpability of the radial artery after occluding the brachial artery by compression]]></category>
		<category><![CDATA[poor compressiblity of the arteries with the cuff.]]></category>
		<category><![CDATA[Pseudohypertension]]></category>
		<category><![CDATA[sphygmomanometer]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3408</guid>
		<description><![CDATA[ Osler&#8217;s sign is seen with:
	a. Pseudohypertension
	b. White coat hypertension
	c. Postural orthostatic tachycardia
	d. Postural hypotension 
Answer: a

Osler&#8217;s sign is the palpability of the radial artery after occluding the brachial artery by compression. This indicates a thickened arterial wall and can cause falsely high blood pressure to be recorded by a sphygmomanometer due to poor compressiblity [...]]]></description>
			<content:encoded><![CDATA[<p> Osler&#8217;s sign is seen with:</p>
<p>	a. Pseudohypertension<br />
	b. White coat hypertension<br />
	c. Postural orthostatic tachycardia<br />
	d. Postural hypotension </p>
<p><strong>Answer: a<br />
</strong></p>
<p>Osler&#8217;s sign is the palpability of the radial artery after occluding the brachial artery by compression. This indicates a thickened arterial wall and can cause falsely high blood pressure to be recorded by a sphygmomanometer due to poor compressiblity of the arteries with the cuff.</p>
]]></content:encoded>
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		</item>
		<item>
		<title>NYHA Class II</title>
		<link>http://www.cardiophile.net/2009/09/nyha-class-ii.html</link>
		<comments>http://www.cardiophile.net/2009/09/nyha-class-ii.html#comments</comments>
		<pubDate>Thu, 24 Sep 2009 15:42:37 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[Goldman specific activity scale]]></category>
		<category><![CDATA[METS]]></category>
		<category><![CDATA[NYHA Class]]></category>
		<category><![CDATA[NYHA Class I]]></category>
		<category><![CDATA[NYHA Class II]]></category>
		<category><![CDATA[NYHA Class III]]></category>
		<category><![CDATA[NYHA Class IV]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3406</guid>
		<description><![CDATA[ NYHA Class II corresponds to the following level on Goldman Specific Activity Scale:
	a. More than 7 METS
	b. 5 &#8211; 7 METS
	c. 2 &#8211; 5 METS
	d. Less than 2 METS 
Answer: b

Goldman specific activity scale (Goldman L et al. Circulation. 1981; 64:1227-34) corresponds to NYHA Class ~ in the following steps:
NYHA Class I : > [...]]]></description>
			<content:encoded><![CDATA[<p> NYHA Class II corresponds to the following level on Goldman Specific Activity Scale:</p>
<p>	a. More than 7 METS<br />
	b. 5 &#8211; 7 METS<br />
	c. 2 &#8211; 5 METS<br />
	d. Less than 2 METS </p>
<p><strong>Answer: b<br />
</strong></p>
<p>Goldman specific activity scale (Goldman L et al. Circulation. 1981; 64:1227-34) corresponds to NYHA Class ~ in the following steps:</p>
<p>NYHA Class I : > 7 METS<br />
NYHA Class II : 5-7 METS<br />
NYHA Class III: 2-5 METS<br />
NYHA Class IV : < 2 METS </p>
]]></content:encoded>
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		</item>
		<item>
		<title>Kussmaul sign</title>
		<link>http://www.cardiophile.net/2009/09/kussmaul-sign.html</link>
		<comments>http://www.cardiophile.net/2009/09/kussmaul-sign.html#comments</comments>
		<pubDate>Thu, 24 Sep 2009 15:40:41 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[conditions reduced right ventricular compliance or right sided volume over load]]></category>
		<category><![CDATA[Kussmaul sign in JVP]]></category>
		<category><![CDATA[Right ventricular infarction]]></category>
		<category><![CDATA[Right ventricular infarction would be the commonest cause]]></category>
		<category><![CDATA[seldom seen in pericardial effusion with or without tamponade]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3404</guid>
		<description><![CDATA[ Kussmaul sign in JVP may seen in all of the following except:
	a. Cardiac tamponade
	b. Right ventricular infarction
	c. Constrictive pericarditis
	d. Pulmonary embolism 
Answer: a

Kussmaul sign occurs in conditions reduced right ventricular compliance or right sided volume over load. It is seldom seen in pericardial effusion with or without tamponade. Right ventricular infarction would be the [...]]]></description>
			<content:encoded><![CDATA[<p> Kussmaul sign in JVP may seen in all of the following except:</p>
<p>	a. Cardiac tamponade<br />
	b. Right ventricular infarction<br />
	c. Constrictive pericarditis<br />
	d. Pulmonary embolism </p>
<p><strong>Answer: a<br />
</strong></p>
<p>Kussmaul sign occurs in conditions reduced right ventricular compliance or right sided volume over load. It is seldom seen in pericardial effusion with or without tamponade. Right ventricular infarction would be the commonest cause considering its frequency compared to other conditions.</p>
]]></content:encoded>
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		</item>
		<item>
		<title>Korotkoff sounds</title>
		<link>http://www.cardiophile.net/2009/09/korotkoff-sounds.html</link>
		<comments>http://www.cardiophile.net/2009/09/korotkoff-sounds.html#comments</comments>
		<pubDate>Thu, 24 Sep 2009 15:37:21 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[Chronic severe aortic regurgitation]]></category>
		<category><![CDATA[Korotkoff sounds may be heard even up to 0 mm Hg in some of these conditions.]]></category>
		<category><![CDATA[Phase 4 of Korotkoff sounds]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3402</guid>
		<description><![CDATA[Phase 4 of Korotkoff sounds are taken into consideration while recording blood pressure in all of the following conditions except:
	a. Pregnancy
	b. Chronic severe aortic regurgitation
	c. Secondary hypertension
	d. Children 
Answer: c

Korotkoff sounds may be heard even up to 0 mm Hg in some of these conditions. 
]]></description>
			<content:encoded><![CDATA[<p>Phase 4 of Korotkoff sounds are taken into consideration while recording blood pressure in all of the following conditions except:</p>
<p>	a. Pregnancy<br />
	b. Chronic severe aortic regurgitation<br />
	c. Secondary hypertension<br />
	d. Children </p>
<p><strong>Answer: c<br />
</strong></p>
<p>Korotkoff sounds may be heard even up to 0 mm Hg in some of these conditions. </p>
]]></content:encoded>
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		</item>
		<item>
		<title>Familial myxoma</title>
		<link>http://www.cardiophile.net/2009/09/familial-myxoma-2.html</link>
		<comments>http://www.cardiophile.net/2009/09/familial-myxoma-2.html#comments</comments>
		<pubDate>Thu, 24 Sep 2009 15:30:27 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[Carney complex]]></category>
		<category><![CDATA[Carney complex constitutes about 10% of cardiac myxomas and is an autosomal dominant disorder]]></category>
		<category><![CDATA[endocrine dysfunction may be associated]]></category>
		<category><![CDATA[Lentiginosis]]></category>
		<category><![CDATA[myxoma]]></category>
		<category><![CDATA[Myxomas may occur at multiple sites]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3396</guid>
		<description><![CDATA[Familial myxoma is part of:
	a. Carney complex
	b. Down syndrome
	c. Holt Oram syndrome
	d. Eisenmenger complex 	
Answer: a
Carney complex constitutes about 10% of cardiac myxomas and is an autosomal dominant disorder. Myxomas may occur at multiple sites and recur even after complete surgical removal. Lentiginosis, hyperpigmentation and endocrine dysfunction may be associated.
]]></description>
			<content:encoded><![CDATA[<p>Familial myxoma is part of:</p>
<p>	a. Carney complex<br />
	b. Down syndrome<br />
	c. Holt Oram syndrome<br />
	d. Eisenmenger complex 	</p>
<p><strong>Answer: a</strong></p>
<p>Carney complex constitutes about 10% of cardiac myxomas and is an autosomal dominant disorder. Myxomas may occur at multiple sites and recur even after complete surgical removal. Lentiginosis, hyperpigmentation and endocrine dysfunction may be associated.</p>
]]></content:encoded>
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		</item>
		<item>
		<title>CMV vs BMV</title>
		<link>http://www.cardiophile.net/2009/09/cmv-vs-bmv-2.html</link>
		<comments>http://www.cardiophile.net/2009/09/cmv-vs-bmv-2.html#comments</comments>
		<pubDate>Thu, 24 Sep 2009 00:32:41 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[balloon mitral valvotomy]]></category>
		<category><![CDATA[BMV]]></category>
		<category><![CDATA[closed mitral valvotomy]]></category>
		<category><![CDATA[CMV]]></category>
		<category><![CDATA[double atrial contour of left atrial enlargement]]></category>
		<category><![CDATA[features of pulmonary arterial hypertension]]></category>
		<category><![CDATA[Left atrial appendage is amputated during closed mitral valvotomy]]></category>
		<category><![CDATA[mitral restenosis]]></category>
		<category><![CDATA[prominent left atrial appendage shadow]]></category>
		<category><![CDATA[pulmonary venous hypertension]]></category>
		<category><![CDATA[restenosis]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3394</guid>
		<description><![CDATA[ Difference in X-ray chest finding between mitral restenosis after closed mitral valvotomy vs restenosis after balloon mitral valvotomy:
a. Absence of double atrial contour of left atrial enlargement
b. Absence of features of pulmonary arterial hypertension
c. Absence of features of pulmonary venous hypertension
d. Absence of a prominent left atrial appendage shadow 
Answer: d
Left atrial appendage is [...]]]></description>
			<content:encoded><![CDATA[<p> Difference in X-ray chest finding between mitral restenosis after closed mitral valvotomy vs restenosis after balloon mitral valvotomy:</p>
<p>a. Absence of double atrial contour of left atrial enlargement<br />
b. Absence of features of pulmonary arterial hypertension<br />
c. Absence of features of pulmonary venous hypertension<br />
d. Absence of a prominent left atrial appendage shadow </p>
<p><strong>Answer: d</strong></p>
<p>Left atrial appendage is amputated during closed mitral valvotomy as it is an important site for thrombus formation. Hence prominence of left atrial appendage shadow is unlikely in mitral restenosis after closed mitral valvotomy.</p>
]]></content:encoded>
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		</item>
		<item>
		<title>Anisosphygmia</title>
		<link>http://www.cardiophile.net/2009/09/anisosphygmia-2.html</link>
		<comments>http://www.cardiophile.net/2009/09/anisosphygmia-2.html#comments</comments>
		<pubDate>Thu, 24 Sep 2009 00:32:02 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[Anisosphygmia may be seen in all of the following except:]]></category>
		<category><![CDATA[Anisosphygmia or difference in the pulse volume / systolic pressure between two sides]]></category>
		<category><![CDATA[aortic dissection]]></category>
		<category><![CDATA[Coanda effect]]></category>
		<category><![CDATA[In supra valvar aortic stenosis it is due to the direction of the jet to the right (Coanda effect)]]></category>
		<category><![CDATA[Supra valvar aortic stenosis]]></category>
		<category><![CDATA[Takayasu arteritis]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3391</guid>
		<description><![CDATA[Anisosphygmia may be seen in all of the following except:
a. Supra valvar aortic stenosis
b. Aortic dissection
c. Takayasu arteritis
d. Aortic regurgitation
Answer: d
Anisosphygmia or difference in the pulse volume / systolic pressure between two sides can occur in aortic dissection, Takayasu arteritis and supra valvar aortic stenosis. In supra valvar aortic stenosis it is due to the [...]]]></description>
			<content:encoded><![CDATA[<p>Anisosphygmia may be seen in all of the following except:</p>
<p>a. Supra valvar aortic stenosis<br />
b. Aortic dissection<br />
c. Takayasu arteritis<br />
d. Aortic regurgitation</p>
<p><strong>Answer: d</strong></p>
<p>Anisosphygmia or difference in the pulse volume / systolic pressure between two sides can occur in aortic dissection, Takayasu arteritis and supra valvar aortic stenosis. In supra valvar aortic stenosis it is due to the direction of the jet to the right (Coanda effect).</p>
]]></content:encoded>
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		</item>
		<item>
		<title>Wenckebach’s AV block</title>
		<link>http://www.cardiophile.net/2009/09/wenckebach%e2%80%99s-av-block.html</link>
		<comments>http://www.cardiophile.net/2009/09/wenckebach%e2%80%99s-av-block.html#comments</comments>
		<pubDate>Wed, 23 Sep 2009 00:59:32 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[dropped QRS]]></category>
		<category><![CDATA[increments in PR interval]]></category>
		<category><![CDATA[increments in PR interval in Wenckebach’s AV block are decremental]]></category>
		<category><![CDATA[Longest RR interval is less than twice the shortest RR interval]]></category>
		<category><![CDATA[Progressive prolongation of PR interval]]></category>
		<category><![CDATA[Progressive shortening of RR interval]]></category>
		<category><![CDATA[Wenckebach’s AV block is characterised by]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3385</guid>
		<description><![CDATA[Wenckebach’s AV block is characterised by all of the following except:
a. Progressive prolongation of PR interval followed by a dropped QRS
b. Progressive shortening of RR interval
c. Incremental increments in PR interval
d. Longest RR interval is less than twice the shortest RR interval 
Answer: c
The increments in PR interval in Wenckebach’s AV block are decremental. This [...]]]></description>
			<content:encoded><![CDATA[<p>Wenckebach’s AV block is characterised by all of the following except:</p>
<p>a. Progressive prolongation of PR interval followed by a dropped QRS<br />
b. Progressive shortening of RR interval<br />
c. Incremental increments in PR interval<br />
d. Longest RR interval is less than twice the shortest RR interval </p>
<p><strong>Answer: c</strong></p>
<p>The increments in PR interval in Wenckebach’s AV block are decremental. This is the reason for the progressive shortening of RR intervals.</p>
]]></content:encoded>
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		<item>
		<title>Venous Corrigan</title>
		<link>http://www.cardiophile.net/2009/09/venous-corrigan.html</link>
		<comments>http://www.cardiophile.net/2009/09/venous-corrigan.html#comments</comments>
		<pubDate>Wed, 23 Sep 2009 00:56:36 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[aortic regurgitation]]></category>
		<category><![CDATA[Corrigan’s sign]]></category>
		<category><![CDATA[CV wave]]></category>
		<category><![CDATA[mitral regurgitation]]></category>
		<category><![CDATA[prominent carotid pulsation seen in aortic regurgitation]]></category>
		<category><![CDATA[prominent V wave]]></category>
		<category><![CDATA[Pulmonary regurgitaton]]></category>
		<category><![CDATA[Tricuspid regurgitation]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3382</guid>
		<description><![CDATA[Venous Corrigan is seen in:
a. Aortic regurgitation
b. Mitral regurgitation
c. Pulmonary regurgitaton
d. Tricuspid regurgitation 
Answer: d
Venous Corrigan is the name given to the prominent V wave (also called the CV wave) seen in the jugular venous pulse in tricuspid regurgitation. Corrigan’s sign is the prominent carotid pulsation seen in aortic regurgitation.
]]></description>
			<content:encoded><![CDATA[<p>Venous Corrigan is seen in:</p>
<p>a. Aortic regurgitation<br />
b. Mitral regurgitation<br />
c. Pulmonary regurgitaton<br />
d. Tricuspid regurgitation </p>
<p><strong>Answer: d</strong></p>
<p>Venous Corrigan is the name given to the prominent V wave (also called the CV wave) seen in the jugular venous pulse in tricuspid regurgitation. Corrigan’s sign is the prominent carotid pulsation seen in aortic regurgitation.</p>
]]></content:encoded>
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		<item>
		<title>Types of right ventricular hypertrophy</title>
		<link>http://www.cardiophile.net/2009/09/types-of-right-ventricular-hypertrophy.html</link>
		<comments>http://www.cardiophile.net/2009/09/types-of-right-ventricular-hypertrophy.html#comments</comments>
		<pubDate>Wed, 23 Sep 2009 00:53:49 +0000</pubDate>
		<dc:creator>Johnson Francis</dc:creator>
				<category><![CDATA[Multiple Choice Questions]]></category>
		<category><![CDATA[Deep S wave in V6]]></category>
		<category><![CDATA[Dominant R wave in V1]]></category>
		<category><![CDATA[LVH]]></category>
		<category><![CDATA[type A right ventricular hypertrophy]]></category>
		<category><![CDATA[type B RVH]]></category>
		<category><![CDATA[type C RVH]]></category>

		<guid isPermaLink="false">http://www.cardiophile.net/?p=3380</guid>
		<description><![CDATA[Type A right ventricular hypertrophy is characterised by:
a. Deep S wave in V1 with tall R wave in V6
b. Dominant R wave in V1 and deep S wave in V6
c. Dominant R wave in V1 without deep S wave in V6
d. Deep S wave in V6 without dominant R wave in V1 	
Answer: b
Dominant R [...]]]></description>
			<content:encoded><![CDATA[<p>Type A right ventricular hypertrophy is characterised by:</p>
<p>a. Deep S wave in V1 with tall R wave in V6<br />
b. Dominant R wave in V1 and deep S wave in V6<br />
c. Dominant R wave in V1 without deep S wave in V6<br />
d. Deep S wave in V6 without dominant R wave in V1 	</p>
<p><strong>Answer: b</strong></p>
<p>Dominant R wave in V1 and deep S wave in V6 (Option B: Type B RVH; Option C: Type C RVH; Option D: LVH)</p>
]]></content:encoded>
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